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måndag 9 april 2018

Desmiini, Sydänproteiineja TRIM32:n kohteento (C_VII) kr.9

https://www.ncbi.nlm.nih.gov/pubmed/22442138
 Kts. TRIM32  kromosomi8 q38.1. , LGMD2H, STM, BBSH (C_VII)
https://www.sciencedirect.com/science/article/pii/S095506741500006X

Desmin related disease: a matter of cell survival failure







Maintenance of the highly organized striated muscle tissue requires a cell-wide dynamic network that through interactions with all vital cell structures, provides an effective mechanochemical integrator of morphology and function, absolutely necessary for intra-cellular and intercellular coordination of all muscle functions. A good candidate for such a system is the desmin intermediate filament cytoskeletal network. Human desmin mutations and post-translational modifications cause disturbance of this network, thus leading to loss of function of both desmin and
of normal function and gain of toxic function are linked to mitochondrial defects, cardiomyocyte eath, muscle degeneration and development of skeletal myopathy and cardiomyopathy.

https://www.ncbi.nlm.nih.gov/gene/1674
Desmiini koodautuu 2 kromosomista.

CSM1; CSM2; CDCD3; CMD1F; LGMD1D; LGMD1E; LGMD2R
Summary
This gene encodes a muscle-specific class III intermediate filament. Homopolymers of this protein form a stable intracytoplasmic filamentous network connecting myofibrils to each other and to the plasma membrane. Mutations in this gene are associated with desmin-related myopathy, a familial cardiac and skeletal myopathy (CSM), and with distal myopathies. [provided by RefSeq, Jul 2008]
Annotation information
Note: We recognize that OMIM uses LGMD1D to refer to the locus on chromosome 6. There are many citations, however, including PubMed 8533766, 15316618, 20682716, and 21376592 which define LGMD1D as the locus on chromosome 7. We are retaining the usage of the HUGO Gene Nomenclature Committee (HGNC). [24 Jul 2017]
Expression
Biased expression in heart (RPKM 2605.8), esophagus (RPKM 953.7) and 6 other tissues See more
Orthologs

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